By Kuan‐Teh Jeang (Eds.)

This quantity covers the newest advances within the mechanisms of pathogenesis of the HIV-1 virus on track cells. Its better half quantity, Advances in Pharmacology fifty six, indicates how new advancements in realizing the virus translate to the scientific surroundings.

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Extra resources for HIV‐1: Molecular Biology and Pathogenesis Viral Mechanisms, Second Edition

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A. (2001). Role of distal zinc finger of nucleocapsid protein in genomic RNA dimerization of human immunodeficiency virus type 1; no role for the palindrome crowning the R‐U5 hairpin. Virology 281, 109–116. Lawrence, D. , Stover, C. , and Summers, M. F. (2003). Structure of the intact stem and bulge of HIV‐1 Psi‐RNA stem‐loop SL1. J. Mol. Biol. 326, 529–542. , Roques, B. , Le Grice, S. , and Darlix, J. L. (1998). Involvement of HIV‐I nucleocapsid protein in the recruitment of reverse transcriptase into nucleoprotein complexes formed in vitro.

The budding process is probably not rigorously exclusive and an RNA present at a greater than background level will likely be captured nonspecifically by Gag/NC to a level partially reflecting its cellular abundance. A. Biology of RNA Capture by the Gag Protein 1. Site of the Packaging Signal In HIV‐1, the major psi region is located downstream of the 50 splice site such that it appears only on the unspliced RNA. In HIV‐2 and SIV it is found upstream of the SD. Cotranslational packaging explains the retained specificity.

And Liang, C. (2003a).

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